How Parkinson’s Dementia and Lewy Body Dementia Are Related Diseases


In a previous article, I talked about how it could be difficult to find information about Lewy Body Dementia research cure options, but how you could look instead for research talking about a Parkinson’s Disease cure instead. If you’re intent on finding a cure for Lewy Body Dementia or at least holistic treatment that will slow the progression of the disease, keep reading what we’ve posted here at AlivenHealthy, but also follow some of the research leads we’ve provided in the Resources or References section at the bottom of the page. We’ll continue adding to the information about this disease over time because it is difficult for people to find holistic treatment for Lewy Body Dementia and the fact is, there are many straightforward treatment options that will slow the course of either Lewy Body Dementia or Parkinson’s Disease. Consider holistic treatments for Multiple Sclerosis, and Amyotrophic Lateral Sclerosis as well if you’ve been diagnosed with Lewy Body Dementia. All of these diseases are related.

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Parkinson’s disease is caused by the loss of dopamine-producing neurons in the substantia nigra. The substantia nigra is a part of the brain that plays a critical role in reward and movement. Lewy Body Dementia shares a number of clinical, neuropsychological, and pathological features with Parkinson’s disease. Scientists who study these two disorders have noted that correctly diagnosing dementia with Lewy bodies is challenging and many patients who show symptoms of this disease may be misdiagnosed as having Parkinson’s [5][6][7]. 


The gold standard for diagnosing dementia with Lewy bodies is a PET scan and single-photo emission computed tomography (SPECT) tracers. Dopamine transporter loss in the brain is the signature feature of Lewy Body Dementia. The loss of dopamine transporters helps to differentiate this disease from other dementias such as Alzheimer’s [6].

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But the boundary between Lewy Body Dementia and Parkinson’s Disease is not clear cut. While researching Parkinson’s disease in the early 1900’s, Friederich, H. Lewy discovered abnormal protein deposits that disrupt the functioning of the brain. When these proteins are found in the substantia nigra of the brain stem, they deplete dopamine and cause symptoms of Parkinson’s disease. But when these Lewy bodies (proteins) are found in other areas of the brain, they deplete acetylcholine levels which causes disturbances in how the patient perceives the world, behaves, and thinks.These two diseases share many symptoms and clinical features. Indeed, if the patient first presents with the symptoms of dementia, the diagnosis is Dementia with Lewy Bodies. Patients who are diagnosed with this form of dementia often develop many of the clinical symptoms of Parkinson’s Disease. On the other hand, if the patient first develops tremors and other classic symptoms and clinical features associated with Parkinson’s Disease, the diagnosis is Parkinson’s [1][6]. 


About 50 years ago, patients who were diagnosed with Parkinson’s Disease had about a 3% chance of developing dementia. Today, the odds of developing dementia after getting diagnosed with Parkinson’s are much higher at 78% [1]. This fact indicates that something has changed in terms of nutrition, the environment, or some other variable over the past 50 years that has quite an impact on the progression of Parkinson’s disease. And in fact, nutrition has changed dramatically in the past fifty years. Some of these important changes include:


  • The end of iodine fortification of bread. Instead iodine is now added to salt, a poor carrier for iodine. Read more here about how a deficiency of iodine can cause cancer and lead to other health problems.
  • The addition of bromine to all commercial bread products as well as to soft drinks (especially citrus drinks like Mountain Dew and Gatorade). Bromine competes with iodine in the body and has a negative impact on the brain and nervous system.
  • The hybridization and genetic modification of fruits to exclude seeds. Seeds contain vitamin B17 (also known as laetrile or amygdalin). 
  • The criminalization of vitamin B17. It is illegal to buy or sell vitamin B17 supplements in the United States because this vitamin has been shown to prevent and in some cases cure all degenerative diseases including cancer. Many Americans eat apricot kernels (20-40 per day throughout the day-not all at once- usually 5 kernels which should be eaten only on an empty stomach. If they’re mixed with food, they become less potent). [4]
  • Inaccessibility of Vitamin B12. Vitamin B12 should be taken sublingually or as a shot. Vitamin B12 supplements taken by mouth are often ineffective. Symptoms of vitamin B12 deficiency are similar to and/or mimic symptoms of dementia.
  • Choline and Phosphatidylcholine – Choline is a precursor of phosphatidylcholine and acetylcholine. Acetylcholine plays a role in Lewy Body Disease as well as Parkinson’s. [1][2][3][5]


Other important changes that might be relevant to the development of Lewy Body Dementia and Parkinson’s Disease:

  • Adding bromine to swimming pools instead of chlorine for water purification. Symptoms of bromine intoxication mimic and include symptoms of dementia. Bromine builds up in the body over time. 
  • The use of bromine as a fire retardant in furnishings and pajamas as well as vehicle interiors. 
  • While mercury amalgam fillings are not used in most of the world, the U.S. has continued to endorse the use of amalgam for dental fillings. Mercury and other heavy metals have been implicated by many studies as a possible cause of various types of dementia including Alzheimer’s and Lewy body disease.


Parkinson’s disease is considered by many experts to be caused by a lack of cellular energy. The web site offers detailed information on a treatment protocol for Parkinson’s and Lewy body dementia. Though this web site appears to focus primarily on treating cancer, the information is relevant and very useful for a variety of diseases. An in-depth discussion about why this is the case is beyond the scope of this book, but suffice it to say that many of the most important alternative treatments for cancer are important alternative treatments for dementia as well because many of these diseases stem from the same root cause. The CancerTutor protocol views these two diseases (cancer and dementia) as stemming from similar causes as well.


Related Posts:

Natural, At-Home Mucuna pruriens and Methylene Blue Treatment for Parkinson’s Disease and Lewy Body Dementia

The Role of the Gut Microbiome and the Vagus Nerve in Parkinson’s, Multiple Sclerosis, Amyotrophic Lateral Sclerosis (Lou Gehrig’s Disease), Lewy Body Dementia and Other Neurological Diseases

The Need for Alternative Treatments of Dementia and Degenerative Neuronal Disease

The Role of Iodine Deficiency in Parkinson’s Disease and Lewy Body Dementia

Gerson Protocol: Juicing Diet and Coffee Enemas

The Role of Bromine In the Development of Parkinson’s Disease, Lewy Body Dementia, Amyotrophic Lateral Sclerosis (Lou Gehrig’s Disease), and Multiple Sclerosis

Lewy Body Dementia, Amyotrophic Lateral Sclerosis, Multiple Sclerosis, and Parkinson’s Disease: The Role of B -Vitamins Including Vitamin B17 (Laetrile)

Pau d’Arco (Lapachol/Tabebuia Impetiginosa) and Its Effects on Parkinson’s Disease, Multiple Sclerosis, Amyotrophic Lateral Sclerosis (Lou Gehrig’s Disease) and Lewy Body Dementia

PEMF (Pulsed Electromagnetic Frequency) Machines to Treat Lewy Body Dementia, Parkinson’s Disease, Multiple Sclerosis, and Amyotrophic Lateral Sclerosis

Melatonin and REM Sleep Behavior Disorder



[1] Olthof, M. R., Brink, E. J., Katan, M. B., Verhoef, P. (2005). Choline supplemented as phosphatidylcholine decreases fasting and postmethionine-loading plasma homocysteine concentrations in healthy men. Retrieved June 22, 2019 from 


[2] Wood, J. L., Allison, R. G. (1982). Effects of consumption of choline and lecithin on neurological and cardiovascular systems. Retrieved June 22, 2019 from


[3] Dr. Axe (2019). What is choline? Retrieved June 22, 2019 from 


[4] Griffin, G. E. (1974). World Without Cancer, 3rd Ed. American Media Inc. 


[5] Barbeau, A. (1978). Emerging treatments: replacement therapy with choline or lecithin in neurological diseases. Retrieved June 22, 2019 from